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...Ellisman MH, Newmeyer DD (2007) Mitochondria frozen with trehalose retain a number of biological functions and preserve outer membrane integrity. Cell |authors=Yamaguchi R, Andreyev A, Murphy AN, Perkins GA, Ellisman MH, Newmeyer DD

|title=Andreyev AY, Kushnareva YE, Starkov AA (2005) Mitochondrial metabolism of reactive o |authors=Andreyev AY, Kushnareva YE, Starkov AA

..., Dykens J, Terkeltaub R (2000) Mitochondrial oxidative phosphorylation is a downstream regulator of nitric oxide effects on chondrocyte matrix synthesi |authors=Johnson K, Jung A, Murphy A, Andreyev A, Dykens J, Terkeltaub R

|title=Divakaruni AS, Hsieh WY, Minarrieta L, Duong TN, Kim KKO, Desousa BR, Andreyev AY, Bowman CE, Caradonna K, Dranka BP, Ferrick DA, Liesa M, Stiles L, Roger ...hors=Divakaruni AS, Hsieh WY, Minarrieta L, Duong TN, Kim KKO, Desousa BR, Andreyev AY, Bowman CE, Caradonna K, Dranka BP, Ferrick DA, Liesa M, Stiles L, Roger

...ntributing to the damage of cellular components. More recent data point to a more physiological role of mitochondrial-produced ROS in mitochondria-cell ...source of ROS [2,3]. However, our research indicates that mitochondria are a ROS buffering system that can maintain the intracellular level of ROS in re

...uh7 cells. Surprisingly, low concentration of etomoxir (~20-40 µM) exerted a ~17% and 13% inhibitory effect on N- and S-linked respiration respectively ...application as specific inhibitor of FAO in respirometry and suggests that a profound characterization of etomoxir toxicity is required.