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Difference between revisions of "Juhasz Laszlo"

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Revision as of 14:17, 12 June 2017

Name JuhĂĄsz LĂĄszlĂł,
Institution
Juhasz L.jpg
Institute of Surgical Research

Faculty of Medicine University of Szeged

Address SzƑkefalvi-Nagy BĂ©la utca 6., 6720
City Szeged
State/Province
Country Hungary
Email jilajila@gmail.com
Weblink
O2k-Network Lab HU Szeged Boros M


Labels:



Publications

 PublishedReference
Tallosy 2021 Sci Rep2021TallĂłsy SP, Poles MZ, Rutai A, Fejes R, JuhĂĄsz L, BuriĂĄn K, SĂłki J, SzabĂł A, Boros M, Kaszaki J (2021) The microbial composition of the initial insult can predict the prognosis of experimental sepsis. Sci Rep 11:22772.
Poles 2021 Front Immunol2021Poles MZ, NĂĄszai A, GulĂĄcsi L, CzakĂł BL, GĂĄl KG, Glenz RJ, Dookhun D, Rutai A, TallĂłsy SP, SzabĂł A, LƑrinczi B, SzatmĂĄri I, FĂŒlöp F, VĂ©csei L, Boros M, JuhĂĄsz L, Kaszaki J (2021) Kynurenic acid and its synthetic derivatives protect against sepsis-associated neutrophil activation and brain mitochondrial dysfunction in rats. Front Immunol 12:717157. https://doi.org/10.3389/fimmu.2021.717157
BEC 2020.1 doi10.26124bec2020-0001.v12020Gnaiger E et al ― MitoEAGLE Task Group (2020) Mitochondrial physiology. Bioenerg Commun 2020.1. https://doi.org/10.26124/bec:2020-0001.v1
Naszai 2019 Sci Rep2019NĂĄszai A, Terhes E, Kaszaki J, Boros M, JuhĂĄsz L (2019) Ca(2+)N it be measured? Detection of extramitochondrial calcium movement with high-resolution FluoRespirometry. Sci Rep 9:19229.
Rutai 2019 Shock2019Rutai A, Fejes R, Juhåsz L, Tallósy SP, Poles MZ, Földesi I, Mészåros AT, Szabó A, Boros M, Kaszaki J (2019) Endothelin A and B receptors: Potential pargets for microcirculatory-mitochondrial therapy in experimental sepsis. Shock 54:87-95.

Abstracts

 PublishedReference
Rutai 2019 Crit Care2019Rutai A, Fejes R, TallĂłsy SZ, Poles M, JuhĂĄsz L, MĂ©szĂĄros A, Boros M, Kaszaki J (2019) Potential role of endothelin receptors in the therapy of experimental sepsis. Crit Care.
Juhasz 2017 MiPschool Obergurgl2017
Juhasz L.jpg
Blockade of N-methyl-D-aspartate receptors improves polymicrobial sepsis-evoked mitochondrial dysfunction in rats.