Difference between revisions of "Krako Jakovljevic 2021 BEC PD"
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|journal=Bioenerg Commun | |journal=Bioenerg Commun | ||
|abstract= | |abstract= | ||
Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to | Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to synthesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinsonās disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression. | ||
|keywords=ATP, mitochondrial homeostasis, mitochondrial respiration, Parkinsonās disease, cellular models of Parkinsonās, qualitative analysis | |keywords=ATP, mitochondrial homeostasis, mitochondrial respiration, Parkinsonās disease, cellular models of Parkinsonās, qualitative analysis |
Revision as of 16:18, 3 November 2021
Krako Jakovljevic N, Ebanks B, Katyal G, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinsonās Disease. Bioenerg Commun 2021.2. doi:10.26124/bec:2021-0002 |
Ā» Published online: 2021-Oct-06
Krako Jakovljevic Nina, Ebanks Brad, Katyal Gunjan, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta (2021-10-06) Bioenerg Commun
Abstract: Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to synthesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinsonās disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression. ā¢ Keywords: ATP, mitochondrial homeostasis, mitochondrial respiration, Parkinsonās disease, cellular models of Parkinsonās, qualitative analysis ā¢ Bioblast editor: Gnaiger E, Cardoso LHD ā¢ O2k-Network Lab: RS Belgrade Lalic NM, UK Nottingham Chakrabarti L, UK Leicester Moisoi N
ORCID: Krako Jakovljevic Nina, Ebanks Brad, Katyal Gunjan, Chakrabarti Lisa, , Markovic Ivanka Moisoi Nicoleta
Preprint
Labels:
Pathology: Parkinson's
Organism: Human, Mouse, Drosophila